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The prolongation of QTc interval in each category of IVCD subjects was entirely secondary to a prolonged depolarization time, as the repolarization intervals were not significantly different from those observed in the control group (F = 0.5, p = NS). All of these intervals were significantly prolonged compared to 430 ± 4.3 msec in the control group.
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Results: A significant difference in the IVCD 2 hours after hemodialysis was seen between the hypertensive and the normotensive groups. Therefore, my preference would be to describe this type of supraventricular QRS morphology as IVCD with LAD (left axis deviation). The QTc (QT interval corrected for heart rate) in subjects with IVCD were 445 ± 6.8 msec (mean ± SEM) in those with LAD, 470 ± 9.1 msec with RBBB, and 489 ± 6.9 msec with LBBB. The IVCD was measured in all groups by ultrasound and Doppler echocardiography to estimate the pulmonary flow and post dialysis plasma atrial naturetic peptide (ANP). Now since there is IVCD as the reason for QRS widening to say in addition there is LAHB to me is a contradiction in terms because that would make for 2 different conduction defects. The observed intervals in IVCD subjects were compared to similar intervals in 33 healthy individuals in whom there was no evidence for intraventricular conduction abnormalities. To evaluate the relative contribution of the depolarization and the repolarization time prolongation to the prolonged QT interval in patients with intraventricular conduction delay (IVCD), the QRS, QT, and JT intervals were measured in 72 subjects with various types of IVCD. The conventional QT interval measurement, however, includes in its measure the cardiac depolarization (QRS) as well as the cardiac repolarization (JT) intervals. A prolonged QT interval is an important prognostic indicator for cardiac arrhythmias and sudden death.